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NRF2 activation can also result in increased xCT accumulation, which eventually results in high GSH dependency of the affected cells

146 However, cell-based or tissue-engineered constructs (either allogeneic or autologous) may eventually succumb to the underlying pathology and degenerate similar to native tissue

Unfortunately, they have many limitations, and their evidence is not nearly as high compared to that of clinical studies (this is further elaborated in the Future Prospects section)

Keywords: N -acetylcysteine, mechanism of action, antioxidant, anti-inflammatory 1

11,15 Additionally, there is cellular interaction between ferroptosis and other cell death modes (Fig