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COPPER DYSREGULATION RELATED TO ATP7B GENETIC ISSUES IN WILSON DISEASE Excessive hepatic copper deposition in Wilson disease results from impaired biliary excretion of excess copper due to dysfunction of the ATP7B gene [20,21,24,27]

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At the same time, endothelial cell senescence also affects the delivery of nutrients and oxygen, leading to metabolic abnormalities and further accelerating organismal aging (Boutouyrie et al., 2021

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