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These ubiquitin modifications stabilize Complex I at the plasma membrane and create a scaffold for the recruitment of downstream signaling molecules, thereby promoting the activation of pro-survival NF-B signaling cascades
doi: 10.1021/acschemneuro.3c00127 214 SaundersN
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Further, these data suggest that at acute time points following Gclc genetic deletion, compensation by NRF1 could be responsible for the dispensable role of NRF2 in GSH-depleted conditions

10.1007/s43032-021-00699-9 62 KalafatE.SukurY
