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[DOI] [PubMed] [Google Scholar] 106.Devries M.C., Hamadeh M.J., Phillips S.M., Tarnopolsky M.A

However, its compelling basic highlights (particular vascular effect, activation of the collateral pathways) [18,19,20,21,22,23,24,25,26,27,28,29,30,31,32,33,34,35,36,37,38,39,40,41] in the most valuable animal models might still be far from the vast clinical evidence obtained in the huge number of clinical trials [1]

6 Therapeutic opportunities targeting the ferroptosis-neuroinflammation axis As inflammation and ferroptosis are both promoters of depression and their bidirectional interaction may exacerbate the progression of depression, drugs that suppress both inflammation and ferroptosis appear to have potential antidepressant effects