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Studies in rats [61] also suggested that both VPA and its unsaturated metabolites inhibit -oxidation through different mechanisms, such as sequestration of CoA-SH and direct inhibition of specific enzymes in the -oxidation sequence by CoA esters, particularly 4-en-VPA-CoA

Mechanistically, hyperoside inhibits cyclin-dependent kinase 6, thereby promoting dephosphorylation and nuclear translocation of transcription factor EB, which in turn upregulates PGC-1 and uncoupling protein 1 to activate thermogenic programs in adipose tissue

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It also helps keep the drug from harming the environment