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FMT treatment exhibited the ability to restore the balance of Clostridium species in the fecal microbiota, normalize hippocampal brain-derived neurotrophic factor expression, and increase glutathione S-transferase levels in the brain (14, 15)

The EMPA-REG and CANVAS trials showed that the SGLT-2i empagliflozin and SGLT-1/2i canagliflozin reduced the risk of MACE in T2D patients with or at high risk of CVD by 14%, while the SGLT-2i dapagliflozin did not significantly reduce the risk of MACE, possibly due to the difference in population (approximately 60% with no established ASCVD) between previous trials and strict exclusion criteria

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[ 2 ] As much of the available evidence is limited, the efficacy, safety profile, and clinical applications of DSIP have not been firmly established