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While SCFA modulation mitigated inflammation and alveolar destruction in a COPD mouse model, it had no effect in the elastase-induced model, suggesting SCFAs might influence COPD pathogenesis through their anti-inflammatory properties, offering potential therapeutic targets (Fig

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4 The reported cases include respiratory injury including acute eosinophilic pneumonia, organizing pneumonia, acute respiratory distress syndrome, and hypersensitivity pneumonitis

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