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Archived from the original on November 13, 2007

The cumulative DNA damage caused by repeated exposure to cigarette smoke carcinogens promotes cell cycle dysregulation and malignant transformations

In a transplantable model of BC the dual CCR1/CCR5 antagonist, Met-CCL5, impeded tumor growth and inhibits the migration of macrophages and lymphocytes into 410.4 tumors, which potentially implicated CCL5 in the facilitation of tumor-promoting macrophage or lymphocyte infiltration

In 1974, Connecticut was the first state to enact restrictions on smoking in restaurants