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Therefore, the upregulation of these genes by p53 possibly serves to sensitise cells to apoptosis by cytotoxic T cells and natural killer (NK) cells, which can express the ligands for death receptors

Mollaoglu G, Guthrie MR, Bohm S, Bragelmann J, Can I, Ballieu PM, et al

Indeed, HCV has a strong ability to induce genomic instability and aneuploidy of hepatocytes, because the ways of its replication had affected the homeostasis of MDM2p53 axis

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We recently linked hyperactivation of Nrf2 through both mutational and transcriptional mechanisms to acquisition and maintenance of resistance to chemotherapy and radiation, the two mainstay treatments for HNSCC as well as for many other solid tumors [18, 19, 51]