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Furthermore, as cisplatin and radiationbased treatment are likely to further select for tumors with extremely high levels of activation[18, 19, 23], ICI-based rescue of conventional treatment failure, or potentiation of conventional chemoradiation strategies may very well be unfeasible at least in a subset of HNSCC [56]

In addition, future studies should focus on the hallmarks of MDM2p53 axis dysfunction in the chronic progressive liver disease that is strongly associated with HCC initiation and development, which will help to diagnose and treat the HCC at vulnerable early stages

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Redox factor 1 (Ref-1) enhances specific DNA binding of p53 by promoting p53 tetramerization