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Description
It has been shown that for thrombosis to take place there must be 1) an abnormal pro-thrombotic activity (e.g., hyperactive platelets[58], increase plasma fibrinogen[59]), 2) abnormal antithrombotic activity (e.g., defect in the inhibitory prostaglandin/PGI 2 s antiplatelet function[60]), 3) an increase in thrombin generation[61], 4) vascular cell damage[62], 6) and fibrinolysis system failure or inhibition[63]

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