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Therefore, a possible interpretation for these findings is that the downregulation of both UCP3 and ANT might be a compensatory mechanism to maintain the proton-motive force and ATP synthesis in a condition where the respiratory complexes protein abundance becomes critically low and chronically elevated levels of 4-HNE or repeated cigarette smoke exposure would otherwise continuously lessen the mitochondrial membrane potential and compromise intracellular ATP levels

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"A large brewing company or fast-food chain certainly wouldn't, and we're no different." In a country where fitness at times appears to be the national religion, smoking has declined markedly
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The control and ASA30 groups were exposed to room air during the exposure process and received a daily oral gavage of tris-base and ASA (30 mg/kg), respectively, for eleven executive days
