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Occupational exposures also do not consider exposure to sensitive subgroups such as people with medical conditions, children, and infants who might be exposed to secondhand e-cigarette emissions, most notably nicotine

Certainly, p53 can mediate HBx protein inactivation through MDM2-dependent ubiquitin degradation and even seven in absentia homolog 1 (Siah-1) protein-dependent proteasomal degradation 66,67

By reactivating mutp53, we can potentially delay the need for surgical interventions, offering a novel strategy of SC prevention that may improve the quality and life expectancy of a population increasingly susceptible to UVR-induced SC

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Cardiovascular disease deserves equal attention