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Potential Suppressive Effect of Nicotine on the Inflammatory Response in Oral Epithelial Cells: An In Vitro Study

In this assay, a single 45 mg dose of non-adjuvanted CoV-RBD121-NP vaccine formulation showed the highest effect

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With regard to the molecular toxic mechanisms of tobacco, oxidative stress or inflammasome activation as well as a chronic activation of aryl hydrocarbon receptor signaling can contribute to premature aging and the development of neoplasms by affecting metabolism, extracellular matrix remodeling, inflammation, pigmentation, DNA repair, and apoptosis 10