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In line with this notion, pharmacologic or genetic suppression of GLP-1R in the lateral septum attenuates the ability of cocaine to activate the mesolimbic dopamine system, as assessed by hyperactivity, reward in the CPP test, and dopamine enhancement in the NAc ( via arachidonic acid and the increased expression of the dopamine transporter within the lateral septum ( Although the vast majority of studies have revealed interactions between cocaine responses and GLP-1R, some studies have reported correlations between the responses to cocaine and the plasma levels of GLP-1

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[5] These situations include moderate to severe nausea and vomiting that develops unusually quickly after injection, unexplained dizziness or feeling faint, unusual pain at the injection site, or symptoms of hypoglycaemia in patients taking other diabetes medications