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An Inflammation-Centric View of Neurological Disease: Beyond the Neuron. Frontiers in Cellular Neuroscience, vol

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In both groups, lixisenatide markedly ameliorated neurological deficit, reduced infarct volume, along with inhibited the expression of oxidative stress parameters (GSH, MDA, NO and catalase enzyme), apoptotic marker (caspase-3), and inflammatory factor (TNF-) in ischemic brains of rats (117)

ACKNOWLEDGMENTS The writing group acknowledges the contribution of Dr Scott Isaacs, who reviewed and provided advice on the Endocrinologic considerations and Adverse events and safety monitoring sections of this manuscript

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