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Mulvihill, [email protected] This article was submitted to Signaling, a section of the journal Frontiers in Cell and Developmental Biology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers
Hansson GK (2005) Inflammation, atherosclerosis, and coronary artery disease
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This finding is consistent with the hypothesis that incretin signalling at GLP-1R and GIPR functionally counterbalances the glucagonergic input under high-glucose conditions

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