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L., Rossing, P., Scott, D., Shamkhalova, M., Bosch-Traberg, H., Syrn, A., & Seino, Y
This is due to expanding cardiovascular outcome data
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Genetic studies suggest an increased ED risk with gliclazide [154], possibly due to SU-mediated inhibition of ATP-sensitive potassium channels essential for prostaglandin E1 (PGE1)- and cAMP-dependent vasodilation [155]
