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Increased understanding of NASH susceptibility and pathogenesis, emerging human multiomics profiling, integration of electronic health record data and modern pharmacology techniques hold enormous promise in delivering a paradigm shift in antifibrotic drug development in NASH

AMPK inhibits inflammation by indirectly inhibiting NFB, a key activator of inflammation [4]

The three major hurdles that prevent faster and vast progress such as less sustainable behavior change, medication side effects, and medication cost