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Recent studies have shown that ferroptosis in chondrocytes aggravates the progress of RA [23], and the cGAS-STING pathway has been proven to positively regulate the ferroptosis in various diseases [24,25,26]
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Originally conceived as a type 2 diabetes therapy rather than a pure obesity drug, DA-1726 was optimized less for maximal appetite suppression and more for metabolic control, which Kim believes may underlie its comparatively better tolerability
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