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DISCUSSION Although the prevalence and clinical impact of AF in people with HFpEF are well recognized, 1,2,9 there are few data exploring this relationship in the context of obesity-related HFpEF

The mechanism of action remains unchanged whether a patient receives compounded or brand-name medication

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Receptor density in cardiovascular and immune tissues is, however, markedly lower than in the pancreas, and the modest physiological increment in GLP-1 produced by DPP-4 inhibition does not appear sufficient to drive the downstream signaling needed for anti-inflammatory, endothelial, or plaque-modifying effects [13], [14], [28]

For those who do not tolerate the medication because of gastrointestinal side effects, the extended release formulation may be more acceptable