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Beyond the microbiota: understanding the role of the enteric nervous system in Parkinsons disease from mice to human

The cause likely involves rapid weight loss mechanics, individual genetic inflammatory response, or GLP-1 receptor activity in muscle and bone tissue rather than a direct drug toxicity

Patients with T2DM experience a significant decline in the incretin function causing delayed and reduced insulin release after glucose administration, impairing glucose tolerance [12,13,14]

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PMC 2925424
