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Here, again, evidence suggests that the neural substrates of alcohol and food craving are largely overlapping, encompassing the ventromedial/orbitofrontal PFC, dorsal and subgenual anterior cingulate cortex, ventral striatum, parietal and temporal areas, cerebellum, and amygdala, with machine-learning classifier algorithms trained on one class of stimuli (e.g., food) reliably predicting craving for the other class (e.g., alcohol), and vice versa [182]

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Although the precise mechanisms of HSD17B13 variants in MASH remain unclear, studies have identified that protein kinase A-mediated phosphorylation of HSD17B13 interacts with adipose triglyceride lipase to promote lipolysis and suppress MASH 66