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But its not the end of the story

Semaglutide's extended half-life of approximately one week contributes to this flexibility

doi:10.1016/j.amjcard.2024.04.041

There are other mechanisms by which circulating incretin mimetics can influence deep brain function without crossing the blood-brain barrier

Because: patients eat less but not necessarily better and often without lifestyle restructuring The real risk We may be creating a new phenotype: Weight loss with metabolic fragility (Less overweight, but not necessarily healthier) The question is no longer: Should we prescribe GLP-1 drugs? But: What happens if patients cant stay on themand what are we not fixing underneath? Bottom line GLP-1 drugs are powerful tools