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However, extrapolating from classic diet trials to GLP-1 pharmacotherapy is imperfect: appetite suppression changes habitual intake patterns, and the degree to which patients spontaneously maintain protein targets and resistance loading during GLP-1 therapy is variable in real-world practice

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Systemic injection of substance P promotes murine Calvarial repair through mobilizing endogenous Mesenchymal stem cells

But its important to keep expectations realistic: this kind of research is mechanistic and early-stage

If you want a broader comparison of how different GLP-1 medications may feel in practice, semaglutide vs tirzepatide side effects is worth a look