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Stimulation of pyloric motility by intraduodenal dextrose in normal subjects
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GLP-1/GIP, GLP-1/glucagon, and even triple agonists are the dominant focus, though the spectrum extends to other mechanisms as wellamylin, GIP antagonism, NK2 receptor agonism, and more, said DiMarchi Looking further ahead, DiMarchi sees potential for oral therapies, including oral peptides
