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Semaglutide, as supplied, is an experimental compound intended as a tool for scientific investigation into the mechanisms of GLP-1 receptor agonism in controlled laboratory settings
Mechanistically, C3G activated the PPAR/--catenin-TCF-4 pathway in STC-1 cells, enhancing PG precursor transcription and GLP-1 synthesis.Inhibiting PPAR/ with GSK0660 blocked this C3G-induced upregulation
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