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3.3 Autophagy dysregulation in comorbidity pathogenesis The comorbidity of diabetes and pancreatic cancer arises from interactions of multiple pathophysiological bases, involving a complex network of genetic susceptibility, metabolic dysregulation, and environmental exposures

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Indeed, GIP administration reduces cell apoptosis and enhances cell mass in animal models [32]

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These findings suggest a change in postsynaptic input

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