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Neurology 101:e2138e2150
Because SCFA production depends on microbiome and diet, GLP-1 medications alone cannot rebuild this pathway
Here, we demonstrate that the effects of cotadutide in reducing body weight and food intake and improving glucose control are predominantly mediated through Glp-1 signalling, whereas its action on the liver to reduce lipid content, drive glycogen flux and improve mitochondrial turnover and function are directly mediated through Gcg signalling
95% CI, 0.380.98) and cardiovascular mortality (RR 0.72
