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Not surprisingly, a specific GLP-1 receptor antagonist, exendin [939] amide, accelerates gastric emptying in individuals without diabetes with proportional rises in plasma glucose and insulin (17), indicating that physiological postprandial concentrations of GLP-1 are sufficient to slow gastric emptying substantially

Side effects are usually temporary and improve through dose titration, smaller meals, and proper hydration

Humanized receptor mouse models express the human targets of these drug candidates, enabling in vivo efficacy and safety evaluation and helping researchers better predict the clinical performance of new obesity and diabetes therapies