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Compared to endogenous incretin hormones which have a very short half-life of a few minutes, exogenous GLP-1 RAs and GIP-RAs have been modified to have a half-life of between 24 h and 7 days depending on the agent [22]

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Since GLP-1R has been localized by immunocytochemistry in mouse aortic smooth muscle cells, endothelial cells, monocytes, and macrophages, both direct and indirect actions of GLP-1 may contribute to the potential reduction of atherogenesis