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These changes were accompanied by a significant reduction in collagen deposition and overall fibrosis severity, suggesting that GLP-1 receptor agonism can modify established fibrogenic signalling cascades within the liver [38]

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Upon treatment with the GLP-1R agonist exendin-4, steatotic HuH7 and HepG2 cells exhibited reduced lipid accumulation compared to vehicle controls, as evidenced by oil red O staining [48]

Available from: _Product_Information/human/002735/WC500165117.pdf

Long-term outcomes beyond the treatment period are rarely reported
