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Enhanced GIP secretion in obesity is associated with biochemical alteration and miRNA contribution to the development of liver steatosis

GIP may enhance weight loss by directly targeting its receptor in the central nervous system to regulate appetite, enhancing GLP-1s anorectic action, and reducing drug-induced nausea in order to improve the tolerability of and compliance to GLP-1 receptor agonists [45]

SCFAs were able to increase the number of L cells in cultured intestinal cells in vitro , leading to an increase in GLP-1 release (Petersen et al., 2014)

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[1] Orforglipron is also chemically different from previous oral GLP-1s