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The authors used non-obese diabetic (NOD) mice that are actually more a model for type 1 diabetes mellitus [11]

Finally, post-approval, we offer ongoing support, including appointment reminders, prescription updates, pharmacy support services, and an easily accessible care team for any treatment-related queries

Proposed mechanisms include enhanced insulinotropic activity, improved adipose tissue handling of nutrients, central effects on appetite and reward pathways, and improved tolerability or pharmacodynamic balance when combined with GLP-1 receptor agonism

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