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Activation of GLP-1R by exendin-4 promoted the osteogenic differentiation, increased bone formation and osteoblast number and inhibited BMSC adipogenic differentiation, but wortmannin (PI3K inhibitor), and GLP-1R sh-RNA partially blocked the exendin-4-induced PI3K/AKT signaling activation and attenuated the facilitating effect of exendin-4 in BMSCs (Meng et al., 2016) (Figure 1)

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bioRxiv 8 May 24 Functional and multi-omic aging rejuvenation with GLP-1R agonism Identifying readily implementable methods that can effectively counteract aging is urgently needed for tackling age-related degenerative disorders