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It is hypothesized that synapse failure and neuronal dysfunction derive from the impact of A oligomer that is intimately related to defective insulin signaling [151,152,153,154]

Danese S, Motte Cd Cde L, Fiocchi C

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however the later-phase clinical trials revealed dose-dependent toxicity and limited brain bioavailability, constraining its translational potential [17, 41, 61, 67, 68].Therefore, co-encapsulation of MB with MO in a nanocarrier system offers a rational strategy for a possible therapeutic synergy, via improving the pharmacokinetic stability, and potentiate disease-modifying efficacy