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Metabolic dysfunction-associated steatotic liver disease (MASLD), also known as nonalcoholic fatty liver disease (NAFLD), has seen its global prevalence surge from 25% between 1990 and 2006 to 38% between 2016 and 2019

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Treatment with GLP-1RAs reduces expression of -smooth muscle actin (-SMA) and collagen type I in HSCs, thereby attenuating fibrogenesis ( /Smad and peroxisome proliferator-activated receptor gamma (PPAR), which regulate HSC activation and extracellular matrix (ECM) synthesis ( In vivo studies conducted on animal models of MASLD/MASH have reveal that GLP-1RAs can reduce liver fibrosis scores and collagen deposition, accompanied by decreased inflammatory infiltration and oxidative stress ( Beyond anti-fibrotic effects, GLP-1RAs promote hepatocyte regeneration and repair

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