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administration of a supposedly physiological dose of GLP-1 (7-36amide) depends on the integrity of abdominal vagal afferents [694], indicating that peripheral GLP-1 can inhibit eating via at least two partly separate pathways

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Since we used no detergent or antigen retrieval reagent in this endosomal co-localization study to avoid destroying the structure of endosomes, in this system, it is very difficult to detect the expression of GLP-1R, which should be present on beta cell membranes

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