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it can centrally inhibit bone formation by binding to leptin receptors in the hypothalamus or locally promote bone formation and inhibit bone resorption by binding to receptors expressed on the surface of osteoblasts [100]
This suggests GLP-1 resistance emerges further downstream
However, there is concern about an increased risk of hypoglycemia and ketotic hyperglycemia
In follow-up from the STEP 1 trial on semaglutide, average weight loss on semaglutide was 17.3% at week 68
2) the intervention consisted of administration of a GLP-1 receptor agonist
