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Implications for employers: Evidence continues to accumulate that GLP-1 medications provide additional clinical benefits as they effectively treat obesity

Mojsovs laboratory was situated within the Endocrine Unit, which would be led by Henry (Hank) Kronenberg from the Fall of 1983 until his retirement more than 35 years later

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Consistent with these results in isolated ventricles suggesting a role for ATP-sensitive K + channels in mediating the effects of GLP-1 during RPc, in mice with Kir6.1 knocked out in pericytes, RPc was not protective against ischaemia/reperfusion injury (Fig

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