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Background: A malfunction in the melanocortin-4 receptor (MC4R) is associated with obesity in rare genetic syndromes
Semaglutide alleviated reserpine induced histopathological and immunohistopathological changes in spinal cord of rats evidenced by a remarkable rise in immuno-expression of cluster of differentiation 163 (CD163) contrary to a significant diminution in CD86 level as compared with reserpine group
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