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Meanwhile, western blot results confirmed that CGA treatment downregulated rotenone-induced phosphorylated alpha-synuclein levels by upregulating PI3K/AKT signaling and inactivating GSK-3 through the release of GLP-1
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in patients with knee osteoarthritis and type 2 diabetes if treatment is sustained for a sufficient period, according to data.The researchers, who published their findings in the Annals of the Rheumatic Diseases, added that the result may be mediated by weight loss.
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