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Supporting insulin release: GLP-1 enhances insulin secretion in response to food, especially when blood sugar levels rise

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Thus, while endocardial endothelial cells represent a plausible target for the antiinflammatory actions of GLP-1 medicines locally in both species, these medicines may also exert cardioprotective effects directly on human cardiomyocytes

GLP-1 RAs inhibit gastric and intestinal motility primarily through actions on myenteric neurons, which modulate gut function via nitrergic and cyclic adenosine monophosphate-dependent pathways

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