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Early clinical trial results have generated significant interest in this triple agonist as a potential next step beyond semaglutide and tirzepatide

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[16] These medications work through multiple complementary mechanisms: they enhance glucose-dependent insulin secretion from pancreatic beta cells, suppress inappropriate glucagon release from alpha cells, slow gastric emptying to reduce postprandial glucose excursions, and act on central nervous system pathways to promote satiety and reduce appetite