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Sanna et al [37] identified human genetic variants that associate with faecal SCFA levels and reported evidence for a potential causal connection between the GMs butyrate production potential (i.e

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Inflammation is the primary pathological mechanism underlying CAD, since is involved in atherosclerotic plaque formation

However, some studies were designed for other purposes 17,18,30,41,42,43,44 for example, prevention of CVD or T2D

Q3: Why should I start with the 0.25mg strength