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They are licensed for diabetes and obesity management, and any blood pressure reduction should be viewed as a beneficial additional effect rather than the primary therapeutic goal

Activation of the modified GIPR and GLP-1R constructs by tirzepatide were confirmed by cAMP accumulation and receptor binding assays, showing similar responses to those of the wild-type (WT) receptors (Supplementary Fig

Studies using exogenous GLP-1 infusion in humans demonstrated reduced energy intake, reduced appetite and decreased brain responses to food anticipation and consumption without direct changes in energy expenditure, and data strongly suggest that body-weight loss ensuing from GLP-1R agonist administration in humans largely reflects reductions in food intake [24, 25]

For GLP-1-related GI symptoms specifically, no published RCT has tested any probiotic in GLP-1 users for the purpose of relieving drug-induced GI effects

JAMA Internal Medicine, 184 (9), 10561064.