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Notably, increased shear stress in diseased vessels of diabetic patients may trigger sustained platelet aggregation independent of soluble agonists as well (Nesbitt et al., 2009), a particularly interesting aspect since major antiplatelet agents act exactly by preventing the release of these agonists

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While there have been conflicting reports within the literature confirming or refuting this latter association, we perform a retrospective cohort analysis and provide a literature review to further review this phenomenon

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