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Description
Neuroprotection of GLP-1/GIP receptor agonist via inhibition of mitochondrial stress by AKT/JNK pathway in a Parkinsons disease model
More specifically, the ARC nucleus of the hypothalamus has been highlighted as one of the main sites mediating this action as chemical lesion of the ARC in rats ablates the anorectic action of GLP-1 (Tang-Christensen et al., 1998)
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Energy begins returning as your body adapts to its new metabolic state
doi: 10.1152/physrev.00034.2006 [DOI] [PubMed] [Google Scholar] 60.Kim W, Egan JM
