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Finally, we hypothesize that Cav-1 may regulate GLP-1 action binding both GLP-1R and -arrestin-1, and modulating the activity of Dipeptidyl Peptidase (DPP)-IV
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In conclusion, this review synthesizes molecular insights and clinical evidence to underscore GLP-1RAs potential as multi-targeted disease-modifying agents, urging further research to translate preclinical promise into safe, effective ocular therapies
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