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Description
Reduced carbohydrate availability constrains glycolytic flux and increases dependence on lipid-derived substrates
Thus, DPP4 inhibition stimulates insulin secretion, suppresses glucagon, and, at least in rodents, promotes cell mass, which enhances islet function
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(van Geenen et al., 2010) found that female Crohns disease patients were at particular risk for Azathioprine-related AP (5.1% vs
